Clear The Air News Tobacco Blog Rotating Header Image

Passive Smoking

Back from the Cold: Russia Confronts Tobacco

You might have missed it over the winter, but Russia achieved an important public health milestone that deserves applause: It enacted a national law that bans smoking in public places and restricts cigarette sales, joining a growing number of countries in making tobacco control a health priority.

http://blogs.worldbank.org/health/back-from-the-cold-russia-confronts-tobacco

The policy victory was a long time coming.

I recall, working in Russia in the mid- and late 2000s, the sense of gloom that prevailed about the poor health conditions of the population. Indeed, in a report that the World Bank prepared in partnership with the Russian Ministry of Health and the World Health Organization (WHO), we documented that Russians were “dying too young,” the main causes driving this phenomenon, and its enormous demographic, social and economic toll.

It was obvious then, as it is now, that a major culprit of the demographic and health decline in Russia is the widespread use of tobacco, which is linked with the country’s high rates of cardiovascular diseases (some of the world’s highest), many cancers and chronic lung diseases. According to the 2008-10 Global Adult Tobacco Survey (GATS), there are nearly 44 million smokers in Russia, and almost every Russian is exposed to secondhand smoke in bars and restaurants. One in three Russians is exposed to smoke in the workplace, and it is estimated that smoking claims 330,000-400,000 Russian lives every year.

While Russia ranks number one in smoking rates among countries surveyed, cigarette smoking is the single most preventable cause of disease and death in the country. And the future disease burden will be influenced by the already high rates of smoking in adolescents and young adults of both sexes in the country.

By the late 2000s, the rising cost to public health from smoking could no longer be ignored. From the highest offices in the Kremlin and the State Duma, to the Ministry of Health, regional governments and civil society, a consensus begun to emerge about the need to combat the tobacco epidemic as part of a broader effort to improve the poor health conditions of the population.

The stark reality underlying this shift was that mortality among Russian men had increased by 60% since 1991 — four to five times higher than the European average — contributing to the precipitous decline in life expectancy among males, to a low of 58 years in 2004 from the peak of 65 years in 1964, while women lived 14 years longer.

Although male life expectancy has risen in recent years to 63 years and life expectancy for women is now 75 years, poor health conditions, alongside declining fertility below replacement levels, have led to a significant decline in the total size of Russia’s population, from 148 million in 1991 to 141 million in 2011. Annual productivity loss from smoking-related premature mortality was estimated to reach US$ 24.7 billion, or more than 3% of the GDP, in 2008. Additional losses from morbidity and health care expenditures related to smoking compound this loss.

The growing political and social momentum toward addressing Russia’s high burden of preventable illness contributed to the decision by the Russian government to ratify in April 2008 the Framework Convention on Tobacco Control (FCTC), a global treaty adopted by the 56th World Health Assembly in 2003, which has now been signed by 168 countries covering close to 90% of the world’s population.

And on February 23, 2013, Russian President Vladimir Putin signed a long-awaited, comprehensive law that will ban smoking in most public places and restrict cigarette sales in the world’s second-largest tobacco market after China (Russia’s tobacco market was estimated to be worth US$22 billion in 2011). The law, which will ban smoking on municipal transport, at railway stations, in lifts, bus stations and administrative buildings, as well as in health facilities and schools beginning June 1, 2013, will come into full force in 2014, covering other venues such as ships, long-distance trains, train platforms, hotels, cafes and restaurants. It will also ban sales of tobacco products at street kiosks, restrict advertising, and set minimum prices for cigarettes, which now cost less than US$2.

On this year’s World Health Day, celebrated on April 7, the Russian people and the global health community should rejoice. The enactment and implementation of this momentous law puts Russia on the right side of public health history. Its measurable benefit in years ahead will be less ill health and disability, countless lives saved, and a more productive and healthy population.

To paraphrase Boris Pasternak in his timeless novel Dr. Zhivago, perhaps “Mother Russia is on the move, she can’t stand still, she’s restless and can’t find rest” — hopefully until the tobacco curse is overcome.

Tobacco in the EU : Exposure to second hand smoke reduced, but still too high, says Commission report

http://europa.eu/rapid/press-release_IP-13-147_en.htm

Tobacco in the EU : Exposure to second hand smoke reduced, but still too high, says Commission report

Other available languages : FR DE DA ES NL IT SV PT FI EL CS ET HU LT LV MT PL SK SL BG RO

European Commission

Press release

Brussels, 22 February 2013

Tobacco in the EU : Exposure to second hand smoke reduced, but still too high, says Commission report

Protection from second hand smoke has improved considerably in the EU, according to a report published by the Commission today. 28% of Europeans were exposed to second hand smoke in bars in 2012 – down from 46% in 2009. The report is based on self-reporting by the 27 Member States, following the 2009 Council Recommendation on Smoke-free Environments (2009/C 296/02), which called upon governments to adopt and implement laws to fully protect their citizens from exposure to tobacco smoke in enclosed public places, workplaces and public transport. The report dispels concerns about smoking bans impacting negatively on the revenues of bars and restaurants, by showing that the economic impact has been limited, neutral and even positive over time. However, the report also illustrates that some Member States are lagging behind, in terms of comprehensive laws protecting public health, and enforcement.

Tonio Borg, European Commissioner for Health and Consumer Policy, said: “The report published today shows that Member States have made steady progress in protecting their citizens from second hand smoke. Citizen’s exposure to smoking, however, still varies widely across the EU and there is a long way to go to make “Smoke Free Europe” a reality. I urge all Member States to step up their efforts to enforce legislation, commend those who have adopted ambitious smoke free laws and urge the others to follow-suit”.

Exposure to second hand tobacco smoke is a wide-spread source of mortality, morbidity and disability in the EU. According to conservative estimates1, more than 70 000 adults in the EU died due to exposure to tobacco smoke in 2002, many of them non-smokers or employees exposed to second hand smoking at their workplaces.

The WHO Framework Convention on Tobacco Control calls on all of its signatories (176 parties) to provide effective “protection from exposure to tobacco smoke in indoor workplaces, public transport, indoor public places, and, as appropriate, other public places”. Guidelines were adopted in 2007 to help parties meet their obligations.

It was against this background that the Council adopted a Council Recommendation on Smoke-free Environments in 2009, calling on Member States to introduce measures to provide effective protection against exposure to second hand smoke no later than November 2012.

Other key findings of the report:

  • All Member States report that they have adopted measures to protect citizens against exposure to tobacco smoke.
  • National measures differ considerably in extent and scope. About half of the Member States have adopted or strengthened their smoke-free legislation since 2009. Many also started earlier.
  • Enforcement seems to be a problem in some Member States. Complex legislation (i.e. legislation with exemptions) is found to be particularly difficult to enforce.
  • The actual exposure rates for EU citizens dropped overall from 2009 to 2012 (e.g. for citizens visiting drinking places the exposure rate dropped from 46% to 28%). There are however very significant differences between Member States.
  • Belgium, Spain and Poland are examples of countries where the adoption of comprehensive legislation led to very significant drops in exposure rates within a short period of time.
  • The positive health effects of smoke-free legislation are immediate and include a reduction in the incidence of heart attacks and improvements in respiratory health.
  • Public support for smoke-free legislation is very high in Europe. A 2009 survey showed that a majority of Europeans are supportive. This is also supported by national surveys which reveal that support increased after introduction of effective measures.

For more information on smoke-free legislation in the EU see:

http://ec.europa.eu/health/tobacco/smoke-free_environments/index_en.htm

Commissioner Borg’s website:

http://ec.europa.eu/commission_2010-2014/borg/index_en.htm

Follow us on Twitter: @EU_Health

Contacts :

Frédéric Vincent (+32 2 298 71 66)

Aikaterini Apostola (+32 2 298 76 24)

Biomarkers of secondhand smoke exposure in automobiles

Download (PDF, 636KB)

Passive Smoking Increases Risk of Severe Dementia, According to Study in China

Web address:
http://www.sciencedaily.com/releases/2013/01/
130109110048.htm

Passive Smoking Increases Risk of Severe Dementia, According to Study in China

Jan. 9, 2013 — An international study by scientists in China, the UK and USA has found a link between passive smoking and syndromes of dementia.

The study of nearly 6,000 people in five provinces in China reveals that people exposed to passive smoking have a significantly increased risk of severe dementia syndromes.

Passive smoking, also known as ‘second-hand’ smoke or environmental tobacco smoke (ETS), is known to cause serious cardiovascular and respiratory diseases, including coronary heart disease and lung cancer. However, until now it has been uncertain whether ETS increases the risk of dementia, mainly due to lack of research. Previous studies have shown an association between ETS and cognitive impairment, but this is the first to find a significant link with dementia syndromes.

The study, published in Occupational and Environmental Medicine, is a collaboration between scientists at King’s College London and Anhui Medical University, China, along with colleagues in the UK and USA.

According to the World Health Organization (WHO), nearly 80 percent of the more than one billion smokers worldwide live in low- and middle-income countries, where the burden of tobacco-related illness and death is heaviest; but only 11 percent of the world’s population are protected by comprehensive smoke-free laws.

China is the largest consumer of tobacco in the world, with 350 million smokers. Since 2006, the Chinese government has actively promoted the introduction of smoke-free environments in hospitals, schools, on public transport and in other public places, but implementation has not been widespread.

Recent data show that the prevalence of passive smoking is still high, with over 50 percent of people exposed to environmental tobacco smoke on a daily basis. China also has the highest number of dementia sufferers in the world, with increasing rates of new cases as the population ages.

Dr Ruoling Chen, senior lecturer in public health from King’s College London, and colleagues interviewed 5,921 people aged over 60 in the rural and urban communities of Anhui, Guangdong, Heilongjiang, Shanghai and Shanxi to characterise their levels of ETS exposure, smoking habits and assess levels of dementia syndromes.

They found that 10 percent of the group had severe dementia syndromes. This was significantly related to exposure level and duration of passive smoking. The associations with severe syndromes were found in people who had never smoked and in former and current smokers.

The data from the Anhui cohort, which were collected at baseline in 2001-03 for dementia syndromes and in the follow up in 2007-08 for ETS exposure and dementia, further excluded the possibility that dementia syndromes caused people to be more exposed to environmental tobacco smoke.

Dr Ruoling Chen, also a visiting professor at Anhui Medical University said: ‘Passive smoking should be considered an important risk factor for severe dementia syndromes, as this study in China shows. Avoiding exposure to ETS may reduce the risk of severe dementia syndromes.

‘China, along with many other countries, now has a significantly aging population, so dementia has a significant impact not only on the patients but on their families and carers. It’s a huge burden on society.’

The findings from this study, together with a second recent study by Chen and colleagues published in Alzheimer’s & Dementia on the links between passive smoking and Alzheimer’s disease, strengthen the case for public health measures to protect people from exposure to environmental tobacco smoke.

‘At present, we know that about 90 percent of the world’s population live in countries without smoke-free public areas. More campaigns against tobacco exposure in the general population will help decrease the risk of severe dementia syndromes and reduce the dementia epidemic worldwide.’

He added: ‘The increased risk of severe dementia syndromes in those exposed to passive smoking is similar to increased risk of coronary heart disease — suggesting that urgent preventive measures should be taken, not just in China but many other countries.’

Share this story on Facebook, Twitter, and Google:

Other social bookmarking and sharing tools:

Share on redditShare on stumbleuponShare on pinterest_shareShare on bloggerShare on diggShare on farkShare on linkedinShare on myspaceShare on newsvine

|18


Story Source:

The above story is reprinted from materials provided by King’s College London.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal Reference:

1.     R. Chen, K. Wilson, Y. Chen, D. Zhang, X. Qin, M. He, Z. Hu, Y. Ma, J. R. Copeland. Association between environmental tobacco smoke exposure and dementia syndromes. Occupational and Environmental Medicine, 2012; 70 (1): 63 DOI: 10.1136/oemed-2012-100785

Top of Form

Need to cite this story in your essay, paper, or report? Use one of the following formats:

APA

MLA

King’s College London (2013, January 9). Passive smoking increases risk of severe dementia, according to study in China. ScienceDaily. Retrieved January 10, 2013, from http://www.sciencedaily.com­ /releases/2013/01/130109110048.htm

Bottom of Form

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Division of Periodontology: Tobacco Use Cessation Program

FACT:  More than 1,324 people die from ETS/SHS inhalation in Hong Kong each year

http://www1.umn.edu/perio/tobacco/secondhandsmoke.html

Secondhand Smoke Facts

Secondhand smoke, also known as passive or environmental tobacco smoke (ETS), is a combination of:

Mainstream smoke: exhaled by smokers

Sidestream smoke: given off by the burning end of a cigarette, cigar, or pipe

Between 70% and 90% of non-smokers in the American population, children and adults, are regularly exposed to secondhand smoke. It is estimated that only 15% of cigarette smoke gets inhaled by the smoker. The remaining 85% lingers in the air for everyone to breathe. If a person spends more than two hours in a room where someone is smoking, the nonsmoker inhales the equivalent of four cigarettes.

Secondhand smoke is the third leading preventable cause of disability and early death (after active smoking and alcohol) in the United States. For every eight smokers who die from smoking, one innocent bystander dies from secondhand smoke.

Secondhand smoke contains over 4000 chemicals including more than 40 cancer causing agents and 200 known poisons.

Secondhand smoke has been classified by the EPA as a Class A carcinogen – a substance known to cause cancer in humans.

Secondhand smoke contains twice as much tar and nicotine per unit volume as does smoke inhaled from a cigarette. It contains 3X as much cancer-causing benzpyrene, 5X as much carbon monoxide, and 50X as much ammonia. Secondhand smoke from pipes and cigars is equally as harmful, if not more so (Mayo Clinic release, Aug 97).

Over the past two decades, medical research has shown that non-smokers suffer many of the diseases of active smoking when they breathe secondhand smoke.

Secondhand smoke causes lung cancer and contributes to the development of heart disease. Never smoking women who live with a smoker have a 91% greater risk of heart disease. They also have twice the risk of dying from lung cancer.

Never-smoking spouses who are exposed to secondhand smoke have about 20% higher death rates for both lung cancer and heart disease.

Secondhand smoke increases heart rate and shortens time to exhaustion. Repeated exposure causes thickening of the walls of the carotid arteries (accelerates atherosclerosis) and damages the lining of these arteries.

When a pregnant woman is exposed to secondhand smoke, the nicotine she ingests is passed on to her unborn baby.

Women who smoke or are exposed to secondhand smoke during pregnancy:

have a higher rate of miscarriges and stillbirths

have an increased risk of low birthweight infants

have children born with decreased lung function

have children with greater risk of sudden infant death syndrome (SIDS)

Children exposed to secondhand smoke are more likely to experience increased frequency of:

asthma, colds, bronchitis, pneumonia, and other lung diseases

middle ear infections

sinus infections

caries in deciduous teeth

Ventilation systems and designated smoking sections do not protect patrons from ETS.

Current estimates of how smoking increases the risk of various diseases are dramatically underestimated because the ill effects of secondhand smoke inhalation are not taken into account.

Particulate mass and polycyclic aromatic hydrocarbons exposure from secondhand smoke in the back seat of a vehicle

Download (PDF, 535KB)

Particulate mass and polycyclic aromatic hydrocarbons exposure from secondhand smoke in the back seat of a vehicle

Download (PDF, 535KB)

3 articles on SFA

Download PDF :

Spain SHS entrances measurements. TC 12 11

UK SHS cars. TC 12 11

US Michigan SFA and cotinine levels. TC 12 11

Tobacco Kills: So what to do in Africa?

The scientific evidence is overwhelming. As Robert Beaglehole and colleagues at the World Health Organization (WHO) pointed out years ago, tobacco is the only consumer product that eventually kills half of its regular users if they follow its manufacturers’ recommendations.

http://blogs.worldbank.org/nasikiliza/tobacco-kills-so-what-to-do-in-africa

Given this dire reality, it is clear that Africa is now at a crossroads. On one hand, the countries in this region have become an attractive and under-tapped market as tougher regulations, high taxes, and greater consumer awareness of the dangers of smoking in developed countries are “closing the door” to tobacco imports and leading to significant drops in consumption. And on the other hand, cigarettes are becoming increasingly affordable as incomes rise in several African countries due to the rapid economic growth of recent years. Indeed, African countries are experiencing the highest increase in the rate of tobacco use amongst developing countries–the number of smokers in sub-Saharan Africa is projected to increase 148 percent by 2030, to 208 million smokers or one-fifth of the total population.
Should African governments and the international community be concerned with this development?

I firmly believe, yes. If the current smoking patterns continue unabated, they will accelerate the growth of tobacco-related non-communicable chronic diseases (e.g., lung cancer, respiratory problems, heart attacks) compounding the already heavy burden imposed by communicable diseases (e.g., HIV/AIDS, tuberculosis, malaria). Besides undermining the health of the population and their productive potential and contributing to lives lost, Africa will be confronted with major health system and funding challenges to deal with these chronic diseases. The magnitude of these challenges is best illustrated by the United States, where cigarette smoking is estimated to cause annually more than 400,000 deaths and about US$200 billion in health-related economic losses, nearly half in direct medical costs.

The good news is that Africa is fighting back. Some 42 African countries have already signed the 2003 WHO Framework Convention on Tobacco Control (FCTC) that binds them to a number of anti-tobacco measures, including high taxes on tobacco products and protecting people from exposure to tobacco smoke.

On June 3-5, 2012, the World Bank, in partnership with the Southern Africa Development Community (SADC), the Ministry of Finance of Botswana, the Bloomberg and Gates Foundations, and WHO, is convening in Gaborone, Botswana, a high level forum “The Economics of Tobacco Control: Taxation and Illicit Trade.” With the participation of delegations from Ministries of Finance, Trade, and Health of 14 SADC member countries and global and regional experts, the aim of the forum is to promote dialogue on best practices in effective design and administration of excise taxes on tobacco as an instrument to promote public health and to share knowledge on the dimensions, causes, and extent of illicit trade of tobacco and strategies to control it.

These topics are critical for helping create a “smoke-free” environment in Africa. Of all the demand-reduction strategies outlined in the FCTC, increasing the retail price of tobacco products through higher excise taxes to make tobacco products less affordable is the single most effective way to decrease consumption and encourage tobacco users to quit. And, contrary to tobacco industry claims, the forum will be geared to show that increased tobacco smuggling does not automatically follow tax increases if governments show committed and strengthened law enforcement to combat smuggling and counterfeiting.

Lung function and exposure to workplace second-hand smoke during exemptions from smoking ban legislation: an exposureeresponse relationship based on indoor PM2.5 and urinary cotinine levels

ABSTRACT
Background The effects of workplace second-hand
smoke (SHS) on lung function remain uncertain because
of a lack of objective measures for SHS exposures.
Objective To determine whether an
exposureeresponse association exists between lung
function and two different markers of SHS based on
indoor fine particulate (PM2.5) and urinary cotinine levels
in non-smoking catering workers.
Design A cross-sectional study during a 1.5-year
exemption of licensed catering premises from smokefree
legislation.
Participants 186 non-smoking catering workers aged
18e65 years in Hong Kong were recruited. A declared
non-smoking status was accepted in workers with
exhaled breath carbon monoxide levels <6 ppm and
urinary cotinine levels <100 ng/ml.
Main outcome measures Lung function measures of
forced expiratory volume in 1s (FEV1 in litres), forced vital
capacity (FVC in litres) and forced expiratory flow as
25e75% of FVC (FEF25e75 in l/s) were recorded.
Results Indoor fine particulate (PM2.5)
concentrations were 4.4 times as high in smoking
premises (267.9 mg/m3) than in non-smoking premises
(60.3 mg/m3) and were strongly associated with the
probability of permitted smoking (R2¼0.99). Smoking
was the dominant source of particulates (R2¼0.66).
Compared with workers exposed to the lowest indoor
PM2.5 stratum (<25 mg/m3), lung function was lower in
the three higher PM2.5 strata (25e75, 75e175,
>175 mg/m3) with FEV1 “0.072 (95% CI “0.123 to
“0.021), “0.078 (95% CI e0.132 to “0.024), “0.101
(95% CI “0.187 to “0.014); FEF25e75 “0.368 (95% CI
“0.660 to “0.077), “0.489 (95% CI “0.799 to
“0.179), “0.597 (95% CI “0.943 to “0.251); and
FEV1/FVC (%) “2.9 (95% CI “4.8 to “1.0), “3.2 (95%
CI “5.1 to “1.4) and “4.4 (95% CI “7.4 to “1.3),
respectively. Urinary cotinine was associated positively
with indoor PM2.5 but negatively with lung function.
Consistently lower values for lung function per unit
increase of indoor PM2.5 were found.
Conclusion Lung function is inversely associated with
workplace SHS. Workplace exemptions and delays in
implementing smoke-free policies and current moves to
relax legislation are a major threat to the health of workers.

ABSTRACTBackground The effects of workplace second-handsmoke (SHS) on lung function remain uncertain becauseof a lack of objective measures for SHS exposures.Objective To determine whether anexposureeresponse association exists between lungfunction and two different markers of SHS based onindoor fine particulate (PM2.5) and urinary cotinine levelsin non-smoking catering workers.Design A cross-sectional study during a 1.5-yearexemption of licensed catering premises from smokefreelegislation.Participants 186 non-smoking catering workers aged18e65 years in Hong Kong were recruited. A declarednon-smoking status was accepted in workers withexhaled breath carbon monoxide levels <6 ppm andurinary cotinine levels <100 ng/ml.Main outcome measures Lung function measures offorced expiratory volume in 1s (FEV1 in litres), forced vitalcapacity (FVC in litres) and forced expiratory flow as25e75% of FVC (FEF25e75 in l/s) were recorded.Results Indoor fine particulate (PM2.5)concentrations were 4.4 times as high in smokingpremises (267.9 mg/m3) than in non-smoking premises(60.3 mg/m3) and were strongly associated with theprobability of permitted smoking (R2¼0.99). Smokingwas the dominant source of particulates (R2¼0.66).Compared with workers exposed to the lowest indoorPM2.5 stratum (<25 mg/m3), lung function was lower inthe three higher PM2.5 strata (25e75, 75e175,>175 mg/m3) with FEV1 “0.072 (95% CI “0.123 to”0.021), “0.078 (95% CI e0.132 to “0.024), “0.101(95% CI “0.187 to “0.014); FEF25e75 “0.368 (95% CI”0.660 to “0.077), “0.489 (95% CI “0.799 to”0.179), “0.597 (95% CI “0.943 to “0.251); andFEV1/FVC (%) “2.9 (95% CI “4.8 to “1.0), “3.2 (95%CI “5.1 to “1.4) and “4.4 (95% CI “7.4 to “1.3),respectively. Urinary cotinine was associated positivelywith indoor PM2.5 but negatively with lung function.Consistently lower values for lung function per unitincrease of indoor PM2.5 were found.Conclusion Lung function is inversely associated withworkplace SHS. Workplace exemptions and delays inimplementing smoke-free policies and current moves torelax legislation are a major threat to the health of workers.

Download PDF : thoraxjnl160291 1..9